On 28th November OxTalks will move to the new Halo platform and will become 'Oxford Events' (full details are available on the Staff Gateway).
There will be an OxTalks freeze beginning on Friday 14th November. This means you will need to publish any of your known events to OxTalks by then as there will be no facility to publish or edit events in that fortnight. During the freeze, all events will be migrated to the new Oxford Events site. It will still be possible to view events on OxTalks during this time.
If you have any questions, please contact halo@digital.ox.ac.uk
Inflammation is a central mechanism in cardiovascular disease pathogenesis, including prominently-studied roles in atherosclerosis and myocardial infarction. Recent clinical trials and GWAS confirm a causal role for inflammation, and for specific inflammatory mechanisms, but the clinical benefit of targeting inflammation remains limited, in part due to inadequate mechanistic understanding that is required to stratify and stage cardiovascular disease. Local opportunities for mechanistic clinical studies include the Oxford Acute Myocardial Infarction (OxAMI) study and the Oxford Heart, Vessels and Fat (HVF) cohort, that have yielded new biomarkers for clinical phenotyping. In experimental models, new mouse models have revealed links between cellular metabolism and redox signalling in inflammation.
