OxTalks will soon move to the new Halo platform and will become 'Oxford Events.' There will be a need for an OxTalks freeze. This was previously planned for Friday 14th November – a new date will be shared as soon as it is available (full details will be available on the Staff Gateway).
In the meantime, the OxTalks site will remain active and events will continue to be published.
If staff have any questions about the Oxford Events launch, please contact halo@digital.ox.ac.uk
Inflammation is a central mechanism in cardiovascular disease pathogenesis, including prominently-studied roles in atherosclerosis and myocardial infarction. Recent clinical trials and GWAS confirm a causal role for inflammation, and for specific inflammatory mechanisms, but the clinical benefit of targeting inflammation remains limited, in part due to inadequate mechanistic understanding that is required to stratify and stage cardiovascular disease. Local opportunities for mechanistic clinical studies include the Oxford Acute Myocardial Infarction (OxAMI) study and the Oxford Heart, Vessels and Fat (HVF) cohort, that have yielded new biomarkers for clinical phenotyping. In experimental models, new mouse models have revealed links between cellular metabolism and redox signalling in inflammation.
