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MAIT cells in host-microbiota interactions
In-person only
The emergence of the MHC I-like gene MR1 in the ancestor of mammals enabled T cells to see new ligands: small metabolites derived either from bile acids or from the microbial riboflavin pathway. MR1 remained highly conserved and monomorphic during evolution, and MR1-restricted T cells (MAIT cells) retained an innate-like program shared across species, indicating non-redundant functions linked to this antigenic specificity. Our lab investigates how MAIT ligands, which are constantly produced by the microbiota, shape MAIT cell development and function.
Date:
30 January 2026, 12:00
Venue:
Kennedy Institute of Rheumatology, Headington OX3 7FY
Venue Details:
Kennedy Lecture Theatre
Speaker:
Dr Francois Legoux (Institut Curie)
Organising department:
Nuffield Department of Orthopaedics, Rheumatology and Musculoskeletal Sciences (NDORMS)
Organisers:
Tess Lawless (Kennedy Institute of Rheumatology),
Katie Roberts (University of Oxford)
Host:
Dr Jethro Johnson (The Kennedy Institute of Rheumatology)
Part of:
Kennedy Institute Seminars
Booking required?:
Not required
Audience:
Members of the University only
Editor:
Katie Roberts
