On 28th November OxTalks will move to the new Halo platform and will become 'Oxford Events' (full details are available on the Staff Gateway).
There will be an OxTalks freeze beginning on Friday 14th November. This means you will need to publish any of your known events to OxTalks by then as there will be no facility to publish or edit events in that fortnight. During the freeze, all events will be migrated to the new Oxford Events site. It will still be possible to view events on OxTalks during this time.
If you have any questions, please contact halo@digital.ox.ac.uk
Alzheimer’s disease (AD) is the leading cause of dementia worldwide, with no cure currently available. AD pathogenesis is primarily driven by cerebral accumulation of β-amyloid (Aβ), a small peptide and proteolytic cleavage product of amyloid-β precursor protein (APP). Elucidating molecular mechanisms that change Aβ levels and APP processing could provide new insights for understanding disease etiology; and development of interventions that attenuate amyloid pathology may provide useful therapeutics for AD.
