On 28th November OxTalks will move to the new Halo platform and will become 'Oxford Events' (full details are available on the Staff Gateway).
There will be an OxTalks freeze beginning on Friday 14th November. This means you will need to publish any of your known events to OxTalks by then as there will be no facility to publish or edit events in that fortnight. During the freeze, all events will be migrated to the new Oxford Events site. It will still be possible to view events on OxTalks during this time.
If you have any questions, please contact halo@digital.ox.ac.uk
We have evidence that the anti-tumour T cell response can, under certain circumstances, drive structural alterations to the tumour microenvironment (TME) which serve to amplify the immune response leading to tumour destruction. Data will be presented which indicates that changes to the TME are widespread and are characterized by the development of specialised blood vessels named high endothelial venules (HEV), re-organization of lymphatic vessels and loss of extracellular matrix (ECM). How these individual features interact and synergise to create conditions favouring immune-mediated cancer destruction will be discussed along with the pivotal role played by antigen-specific T cells.
