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Mitochondria undergo stress induced hyperfusion to adapt their metabolic function and safeguard cellular survival during starvation.
The mechanisms that regulate the underlying cell-biological program are however largely unknown.
In this talk I am going to discuss how lipid transfer at contact sites between mitochondria and lipid droplets controls metabolic remodelling and promotes cell survival.
Specifically I will explain how the lipid transfer protein mitoguardin 2 couples these fundamental aspects of cell physiology.
